Our FANTOM program is dedicated to spreading the fruits of our research far and wide. Early career researchers will engage in diverse outreach activities, including collaborative efforts with the ‘Naked Scientists’ and ‘Open Science,’ providing them with comprehensive training in communication skills. They’ll participate in science festivals, like the Cambridge University Science Festival, where they’ll lead engaging activities to demystify topics such as immune system cancers. Through social media, particularly Twitter, and interactions with the press, they’ll share their findings with both professional and lay audiences. All dissemination efforts will recognize the support received from the European Union’s Horizon 2021 Marie Skłodowska-Curie Doctoral Training Networks.

Will Shepheard (UKE) about his research in podcast series
In my PhD work we are investigating a rare type of T cell lymphoma occuring mostly in children and young adults. We are investigating what happens when a patient stops responding to therapy

Katarina Mišura – PODcast session
In my PhD work we are investigating a rare type of T cell lymphoma occuring mostly in children and young adults. We are investigating what happens when a patient stops responding to therapy

Gerardo Enrique Abarca Ríos – PODcast session
I study a type of blood cancer where the cancer cells depend on a broken signal inside them that tells them to keep growing all the time. There are special drugs designed to block that signal

Nicola Mora – Academic secondment at Klinikum rechts der Isar Technische Universität München (TUM)
Thanks to Professor Lena Illert and my FANTOM colleague Magdalena Kršic, I implemented 2 multiplex immunostaining (6 panel staining) to my ongoing research.

Magdalena Kršić – Academic secondment at the Medical University of Vienna (MUW)
In January 2026, I had the opportunity to begin the new year in a different academic environment through a one-month secondment at the Medical University of Vienna (MUW).

Julia Montague – PODcast session
Some cancers, like anaplastic large-cell lymphoma (ALCL), are driven by a gene called ALK that gets rearranged, causing cancer cells to receive signals to grow more than they should.
